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1 The antiinflammatory drug sulfasalazine inhibits tumor necrosis factor alpha expression in macrophages by inducing apoptosis
article 2000    
Author: Rodenburg, R.J.T. · Ganga, A. · Lent, P.L.E.M. van · Putte, L.B.A. van de · Venrooij, W.J. van
Keywords: Nutrition · Medicine · Geneeskunde · Pharmacology · Toxicology · Caspase 8 · Salazosulfapyridine · Tumor necrosis factor alpha · Animal cell · Antiinflammatory activity · Apoptosis · Crohn disease · Cytokine production · Drug mechanism · Macrophage · Mouse · Nonhuman · Priority journal · Protein expression · Rheumatoid arthritis · Adenosine · Animals · Anti-Inflammatory Agents, Non-Steroidal · Apoptosis · Caspases · Cells, Cultured · Cysteine Proteinase Inhibitors · Humans · Macrophage Activation · Macrophages · Macrophages, Peritoneal · Methotrexate · Mice · Mice, Inbred C57BL · Sulfasalazine · Tumor Necrosis Fac
[Abstract]

2 Bone turnover, joint damage and bone mineral density in early rheumatoid arthritis treated with combination therapy including high-dose prednisolone
article 2001    
Author: Verhoeven, A.C. · Boers, M. · Koppele, J.M. te · Laan, W.H. van der · Markusse, H.M.Geusens, P. · Linden, S. van der
Keywords: Health Biology · Biomedical Research · Adult · Aged · Amino Acids · Anti-Inflammatory Agents · Anti-Inflammatory Agents, Non-Steroidal · Antirheumatic Agents · Arthritis, Rheumatoid · Bone Density · Bone Remodeling · Collagen · Cross-Linking Reagents · Drug Therapy, Combination · Female · Humans · Joints · Male · Methotrexate · Middle Aged · Postmenopause · Prednisolone · Regression Analysis · Sulfasalazine · Bone mineral density · Collagen crosslinks · Deoxypyridinoline · Early rheumatoid arthritis · Glucocorticoids · Outcomes · Pyridinoline
[Abstract]

3 On the assessment of adverse drug reactions from spontaneous reporting systems: The influence of under-reporting on odds ratios
article 2002    
Author: Heijden, P.G.M. van der · Puijenbroek, E.P. van · Buuren, S. van · Hofstede, J.W. van der
Keywords: Health · Pharmacovigilance · Spontaneous reporting system · Under-reporting · Antifungal agent · Antineoplastic agent · Diclofenac · Diuretic agent · Nonsteroid antiinflammatory agent · Terbinafine · Congestive heart failure · Controlled study · Covariance · Drug hypersensitivity · Drug potentiation · Drug surveillance program · Hair loss · Information processing · Risk assessment · Sex difference · Adverse Drug Reaction Reporting Systems · Anaphylaxis · Anti-Inflammatory Agents, Non-Steroidal · Diclofenac · Diuretics · Drug Interactions · Female · Heart Failure, Congestive · Humans · Male · Middle Aged · Odds Ratio · Product Surveillance, Postmarketing · Sex Factors
[Abstract]

4 Release of 5-aminosalicylate from an MMX mesalamine tablet during transit through a simulated gastrointestinal tract system
article 2007    
Author: Tenjarla, S. · Romasanta, V. · Zeijdner, E. · Villa, R. · Moro, L.
Keywords: Biology · Biomedical Research · 5-Aminosalicylate · Bioaccessibility · Gastrointestinal system · In vitro · Mesalamine · MultiMatrix System · Ulcerative colitis · mesalazine · mezavant · nonsteroid antiinflammatory agent · adult · article · controlled study · dialysis · drug clearance · drug coating · drug release · gastrointestinal transit · human · human experiment · in vitro study · normal human · tablet formulation · upper gastrointestinal tract · biological model · chemistry · delayed release formulation · gastrointestinal tract · kinetics · pH · pharmaceutics · Anti-Inflammatory Agents, Non-Steroidal · Delayed-Action Preparations · Gastrointestinal Tract · Hydrogen-Ion Concentration · Kinetics · Mesalamine · Models, Biological · Technology, Pharmaceutical
[Abstract]

5 Stereoselectivity at the B2-adrenoceptor on macrophages is a major determinant of the anti-inflammatory effects of B2-agonists
article 2000    
Author: Izeboud, C.A. · Vermeulen, R.M. · Zwart, A. · Vos, H.P. · Miert, A.S.J.P.A.M. van · Witkamp, R.F.
Keywords: β-Adrenoceptor · Stereoselectivity · 3 isopropylamino 1 (7 methyl 4 indanyloxy) 2 butanol · 4 (3 tert butylamino 2 hydroxypropoxy) 2 benzimidazolone · Aprotinin · Benzylsulfonyl fluoride · Beta 2 adrenergic receptor · Beta 2 adrenergic receptor blocking agent · Beta 2 adrenergic receptor stimulating agent · Carmoterol · cyclic AMP · Interleukin 10 · Lipopolysaccharide · Tumor necrosis factor alpha · Xamoterol · Beta adrenergic receptor stimulating agent · Nonsteroid antiinflammatory agent · Antiinflammatory activity · Stereochemistry · Cell culture · Cell membrane · Drug antagonism · Metabolism · Radioassay · Signal transduction · Stereoisomerism · Adrenergic beta-Agonists · Anti-Inflammatory Agents, Non-Steroidal · Cell Membrane · Cells, Cultured · Cyclic AMP · Humans · Interleukin-10 · Lipopolysaccharides · Macrophages · Radioligand Assay · Receptors, Adrenergic, beta-2 · Signal Transduction · Stereoisomerism · Tumor Necrosis Factor-alpha
[Abstract]

6 Inhibition of tolbutamide 4-methylhydroxylation by a series of non-steroidal anti-inflammatory drugs in V79-NH cells expressing human cytochrome P4502C10
article 1996    
Author: Kappers, W.A. · Groene, E.M. de · Kleij, L.A. · Witkamp, R.F. · Zweers-Zeilmaker, W.M. · Feron, V.J. · Horbach, G.J.
Keywords: Toxicology · CYP2C9 protein, human · cytochrome P450 · diclofenac · enzyme inhibitor · flurbiprofen · ketoprofen · mixed function oxidase · nonsteroid antiinflammatory agent · phenylbutazone · sulfaphenazole · tolbutamide · tolbutamide 4 hydroxylase · tolbutamide 4-hydroxylase · unspecific monooxygenase · animal · article · cell line · Cricetulus · drug interaction · gene expression · genetic transfection · genetics · hamster · human · hydroxylation · lung · metabolism · methylation · Animals · Anti-Inflammatory Agents, Non-Steroidal · Aryl Hydrocarbon Hydroxylases · Cell Line · Cricetinae · Cricetulus · Cytochrome P-450 Enzyme System · Diclofenac · Drug Interactions · Enzyme Inhibitors · Flurbiprofen · Gene Expression · Humans · Hydroxylation · Ketoprofen · Lung · Methylation · Mixed Function Oxygenases · Phenylbutazone · Sulfaphenazole · Tolbutamide · Transfection
[Abstract]

7 Thalidomide inhibits UVB-induced mouse keratinocyte apoptosis by both TNF-α-dependent and TNF-α-independent pathways
article 2003    
Author: Lu, K.Q. · Brenneman, S. · Burns Jr., R. · Vink, A. · Gaines, E. · Haake, A. · Gaspari, A.
Keywords: Toxicology Biology · Toxicology and Applied Pharmacology · α-N-phthalimido-glutarimide · Mouse model · Photoprotection · Sunburn cell · Ultraviolet light · antiinflammatory agent · apoptosis inhibitor · cell DNA · cyclobutane derivative · cytokine · dimer · drug vehicle · messenger RNA · pyrimidine derivative · recombinant tumor necrosis factor alpha · thalidomide · tumor necrosis factor alpha · animal cell · animal experiment · animal model · animal tissue · antiinflammatory activity · apoptosis · article · biosynthesis · bolus injection · cell assay · cell death · controlled study · cytokine production · DNA damage · dose response · drug blood level · drug mechanism · drug megadose · female · histopathology · hypothesis · immunostimulation · in vitro study · keratinocyte · low drug dose · monocyte · mouse · newborn · nick end labeling · nonhuman · photosensitivity disorder · priority journal · radiation dose · radiation exposure · radiation injury · ribonuclease protection assay · skin · skin biopsy · skin lupus erythematosus · steady state · sunburn · ultraviolet B radiation · Animals · Anti-Inflammatory Agents, Non-Steroidal · Apoptosis · Disease Models, Animal · Dose-Response Relationship, Drug · Erythema · Female · Injections, Intravenous · Keratinocytes · Mice · Mice, Inbred BALB C · RNA, Messenger · Thalidomide · Tumor Necrosis Factor-alpha · Ultraviolet Rays
[Abstract]

Search results also available in MS Excel format.

Showing 1 to 7 of 7 found. | Sort by date